iv max-CARDIO
actions
- Reduces Plaque
- Reduces Inflammation
- Reduces Oxidative Stress
HELPS
- High Blood Pressure
- High Cholesterol
- Heart Disease
FREQUENCY
- Tailored to you. Commonly once a week until therapeutic results are obtained (4-8 treatments)
- Reassessment and then maintenance monthly.
HOW DOES THE IV MAX-CARDIO WORK?
Nine in ten Canadians lead a lifestyle that exposes them to at least one key risk of cardiovascular disease. Almost 80% of premature heart disease is preventable through healthier lifestyle choices. A healthy heart requires optimal lifestyle, stress management, sleep, blood sugar, reduced inflammation, and nutrients. This infusion provides the nutrients needed to reduce atheroscerlosis, plaque formation, oxidative damage, inflammation, and promote optimal heart health.
faqs
Ingredients: B Vitamins, Vitamin C, Calcium, Copper, Chromium, Magnesium, Selenium, Zinc, L-Carnitine, Taurine
- B Vitamins help support heart health collectively. Vitamin B3 (niacin) may decrease the production of Lp(a) in the liver 1,2
- Vitamins B6, Folic acid and B12, reduce homocysteine which correlate strongly with the risk of cardiovascular disease.3-5
- Vitamin C along with the amino acids proline and lysine- are essential for the formation of healthy collagen.
- Vitamin C protects the integrity of the blood vessels.6-9
- Calcium: Is required by the heart to contract and pump out blood to the body, it regulates blood pressure and is necessary for blood clotting.
- Magnesium helps to regulate blood pressure, blood sugar, and lipid peroxidation.10
- Selenium is an antioxidant that reduces oxidative damage, inflammation, and increases glutathione; observational studies found a 50% increase in blood selenium levels was associated with a 24% reduction in the risk of heart disease.11
- Zinc may protect the heart muscle from oxidative stress.12
- L-Carnitine the active form of dietary carnitine and a driver of TMAO, is emerging as a target for CVD prevention and treatment, because it has an important role in the oxidation of fatty acids and cardiac energy metabolism13
- Taurine may reduce your risk of cardiovascular disease; research shows a link between higher taurine levels and significantly lower rates of death from heart disease, as well as reduced cholesterol and blood pressure14
OPTIONAL ADD ONS
- IM Fuel D: Vitamin D deficiency has been associated with increased blood pressure, obesity, diabetes mellitus, stroke, congestive heart failure, and metabolic syndrome.15,16
- IV Fuel G Push: Glutathione is a master antioxidant that protects the lining of the arteries, stabilizes platelets, and reduces inflammation, oxidation of fats, and circulating cholesterol.17
B vitamins to reduce homocysteine and energize with chromium to stabilize blood sugar.
Ingredients: Extra B Vitamins, Hydroxy & Methyl B12 (active B12’s), 5-MTHF (active folate), Chromium
- Jafri H, Karas RH, Kuvin JT. Effects of Niacin on LDL Particle Number: Niacin & LDL Particle Number: Niacin & LDL-P. Clin Lipidology. 2009;4(5):565-571\
- Nordestgaard BG, Chapman MJ, Ray K, et al, European Atherosclerosis Society Consensus Panel. Lipoprotein(a) as a cardiovascular risk factor: current status. Eur Heart J. 2010;31:2844-2853.
- Folsom AR, Nieto FJ, McGovern PG, Tsai MY, Malinow MR, et al. (1998) Prospective study of coronary heart disease incidence in relation to fasting total homocysteine, related genetic polymorphisms, and B vitamins: the Atherosclerosis Risk in Communities (ARIC) study. Circulation 98: 204–210.
- Verhoef P, Kok FJ, Kruyssen DA, Schouten EG, Witteman JC, et al. (1997) Plasma total homocysteine, B vitamins, and risk of atherosclerosis. Arterioscler Thromb Vasc Biol 17: 985–995.
- Zhang C, Wang SY, Qin YY, et al, Association between B Vitamins Supplementation and Risk of Cardiovascular Outcomes: A Cumulative Meta-Analysis of Randomized Controlled Trials, PLoS One. 2014; 9(9):e107060
- Carr AC, Frei B. Toward a new recommended dietary allowance for vitamin C based on antioxidant and health effects in humans. American Journal of Clinical Nutrition 1999;69(6):1086-1107.
- Simon JA, Hudes ES. Serum ascorbic acid and gallbladder disease prevalence among US adults: the Third National Health and Nutrition Examination Survey (NHANES III). Arch Intern Med. 2000;160(7):931-936.
- Stephen R, Utecht T. Scurvy identified in the emergency department: a case report. Journal of Emerg Med. 2001;21(3):235-237.
- Cameron E, Pauling L. Supplemental ascorbate in the supportive treatment of cancer: Prolongation of survival times in terminal human cancer. Proc Natl Acad Sci U S A. 1976;73(10):3685-3689
- DiNicolantoni JJ, Liu J, and O’Keefe JH, Magnesium for the prevention and treatment of cardiovascular disease, Open Heart, July 2018, doi 10.1136/openhrt-2018-000775
- Flores-Mateo G, Navas-Acien A, Pastor-Barriuso R, et al, Selenium and coronary heart disease: a meta-analysis, Am J Clin Nutr. 2006 Oct;84(4):762-73
- Brugger D, Windisch WM. Short-Term Subclinical Zinc Deficiency in Weaned Piglets Affects Cardiac Redox Metabolism and Zinc Concentration. The Journal of Nutrition, 2017; 147(4):521
- Klezovitch O; Edelstein C; Scanu AM. Evidence that the fibrinogen binding domain of Apo(a) is outside the lysine binding site of kringle IV-10: a study involving naturally occurring lysine binding defective lipoprotein(a) phenotypes. J Clin Invest 1996 Jul 1;98(1):185-91.
- Boonmark NW; Lou XJ; Yang ZJ; Schwartz K; Zhang JL; Rubin EM; Lawn RM. Modification of apolipoprotein(a) lysine binding site reduces atherosclerosis in transgenic mice. J Clin Invest 1997 Aug 1;100(3):558-64.
- Phillips J; Roberts G; Bolger C; el Baghdady A; Bouchier-Hayes D; Farrell M; Collins P. Lipoprotein (a): a potential biological marker for unruptured intracranial aneurysms. Neurosurgery 1997 May;40(5):1112-5; discussion 1115-7.
- Stubbs P; Seed M; Moseley D; O’Connor B; Collinson P; Noble M. A prospective study of the role of lipoprotein(a) in the pathogenesis of unstable angina. Eur Heart J 1997 Apr;18(4):603-7.
- Shinozaki K; Kambayashi J; Kawasaki T; Uemura Y; Sakon M; Shiba E; Shibuya T; Nakamura T; Mori T. The long-term effect of eicosapentaenoic acid on serum levels of lipoprotein (a) and lipids in patients with vascular disease. J Atheroscler Thromb 1996;2(2):107-9.
- McCully KS, Homocysteine metabolism in scurvy, growth, and arteriosclerosis. Nature 1971;231:391-392.
- Pauling L, Rath M. Pro. Nat. Acad. Sci USA, Vol 87, pp 9388-9390, Dec 1990.
- Marcoux C; Lussier-Cacan S; Davignon J; Cohn JS. Association of Lp(a) rather than integrally-bound apo(a) with triglyceride-rich lipoproteins of human subjects. Biochim Biophys Acta 1997 Jun 23;1346(3):261-74.
- Arsenal D, Hassan S, Reyna SV, Schafer AI, Durante W. Transforming growth factor-b1 stimulates vascular smooth muscle cell L-proline transport by inducing system A amino acid transporter 2 (SAT2) gene expression. Biochem. J. (2001) 360, (507-512)
- White AL; Lanford RE. Cell surface assembly of lipoprotein(a) in primary cultures of baboon hepatocytes. J Biol Chem 1994 Nov 18;269(46):28716-23.
- Murakami S, Taurine and atherosclerosis, Amino Acids. 2014. Jan;46(1):73-80
- Wang TJ et al. Vitamin D and cardiovascular disease risk. Curr Opin Clin Nutr Metab Care. 2008 Jan;11(1):7-12
- Moyad MA. Vitamin D: A Rapid Review. Dermatology Nursing 2009;21(1)
- Lang CA, The Impact of Glutathione on Health and Longevity, J of Anti-Aging Medicine, July 2004, 4 (2)
- Ayers J, Cook J, Koenig RA, et al, Recent Developments in the Role of Coenzyme Q10 for Coronary Heart Disease: a Systematic Review, Curr Atheroscler Rep. 2018 May 16;20(6):29